After the bad news about BVS857 trial, I searched the literature again and I, too, found this publication. And it gave me some new hopes. Let me underline a few points in the study.
I found momentous the following sentences:
"...both type I and type II fibers were significantly reduced in size in exercised AR113Q mice, whereas WT muscle showed the opposite effect." WT means 'wild type'=healthy. So here is the reason of our common experience that our muscles need really long time to regenerate after a hard exercise: our muscles tolerate badly the overuse and we must exercise really carefully.
Another important message of the article: "These findings add to a growing appreciation of non-neuronal phenotypes in this disorder, and raise the possibility that targeting metabolic alterations may alleviate disease manifestations. In support of this hypothesis is recent work demonstrating that feeding mice a high fat diet diminishes aspects of the SBMA phenotype (Rocchi et al., 2016)." Shortly: trying the fat rich diet is a possibility, however, you have to consider your body weight as well. You have to consult your physician first, of course, but, theoretically, it may help. (As a Hungarian I am lucky because the Hungarian food is fat-rich...)
And finally the most important (IMHO): "we speculate that targeting the metabolic phenotype triggered by expression of the polyQ AR may be particularly effective in rescuing non-neuronal manifestations of the disease, including important aspects of skeletal muscle dysfunction." It gives us hope because the non-neuronal targets are easier to reach with drugs, and if you look at the successful Nusinersen-trial, they used the same gene silencing technic as in this animal experiments. I feel the solution is pretty near.